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10.1073/pnas.1119658109 73 MorrisonL

Figure 6 This observation leads us to conclude that vitamin B 12 deficiency must have a secondary effect of inhibiting CBS activity, which in turn inhibits the flux of Hcy converting into cystathionine and protects GSH from changes in Hcy

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By contrast, in diseases where there are defects after the stage of autophagosome formation, for instance in AD and LSDs, then autophagosomes may accumulate unproductively and cause toxicity especially if autophagosome formation is boosted as part of positive feedback loops aimed to promote clearance of accumulating pathogenic substrates